It was reported by a study that was carried out by researchers from Washington University that slow clearance of amyloid protein in brain can take place through Alzheimer's disease.

There was a research conducted in which 12 people with no dementia and 12 people with mild Alzheimer's disease were compared the way their brains produced and cleared a protein called amyloid beta.

Amyloid beta is normally cleared after it is released in the brain, but in Alzheimer's disease cell death is caused by the protein that gets accumulated in the brain.

There has previously been speculation that Alzheimer's disease is caused by an imbalance of amyloid production and clearance.

But it was noted this time that the protein was cleared by 30 per cent slower by people with Alzheimer's disease as compared to unaffected individuals. This suggests that in Alzheimer's disease a potential reason for the accumulation of protein in the brain is slower clearance of the protein, rather than overproduction.

It was however stated by the researchers that it was difficult to figure out if impaired amyloid beta clearance is a cause or consequence of Alzheimer's disease.

However, clear targets for future research is provided by this exciting research that highlights the importance of amyloid beta clearance in Alzheimer's disease.